Wednesday, 17 July 2024
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Researchers uncover the causes of obesity

  • Fructose may be the main cause of obesity, according to a study.
  • The research indicates that the body can also produce fructose from carbohydrates, specifically glucose.
  • Fruits contain a lot of fructose, which greatly inhibits the release of energy.

Fructose, which is present in table sugar and high fructose corn syrup, maybe the main cause of obesity, according to a study by Richard Johnson of the University of Colorado Anschutz Medical Campus.

The research indicates that the body can also produce fructose from carbohydrates, specifically glucose. According to nutritional experts, this finding runs counter to earlier discussions regarding the root cause of obesity.

Causes of obesity

The “fructose survival hypothesis” postulates that the carbohydrate-insulin model, which attributes weight gain to carbohydrates, and the body’s energy balance theory, which contends that excess food, primarily fat, causes obesity, are incompatible.

According to the “fructose survival hypothesis,” fructose causes the body to enter a low-power mode, which reduces appetite, while fatty foods become the main source of calories that lead to weight gain.

The World Health Organization (WHO) estimates that over 1 billion people worldwide are obese, and obesity is a recognized risk factor for several diseases.

According to a recent study that was published in the scientific journal Obesity, there is a single true driver—fructose—that unites all of the theories about what causes obesity, suggesting that these theories are not mutually exclusive.

Fruits contain a lot of fructose, which greatly inhibits the release of energy. Consuming foods high in fructose prevents fat storage from storing active energy, maintaining low levels of active energy.

According to this theory, obesity is a low-energy state, and fructose is the mechanism that directs active energy replacement toward fat storage, indicating that fructose is the primary cause of energy imbalance.

Although further investigation is required to confirm this unifying theory, this is a promising beginning toward the identification of more focused preventative measures for obesity and the management of associated metabolic imbalances.

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